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Involves the formation of a stable fibrin clot through a cascade of proteolytic enzymes. Key players are coagulation factors, which are mostly plasma proteins.

A blood glycoprotein involved in hemostasis, promoting platelet adhesion to the subendothelium after vascular injury and carrying factor VIII.

Activation process of platelets that results in shape change, release of granules, and expression of fibrinogen receptors, leading to aggregation.

Two converging pathways within the coagulation cascade. The intrinsic pathway is activated by internal damage, whereas the extrinsic pathway is activated by external trauma.

The initial response to vascular injury involving vasoconstriction and platelet plug formation. Key factors include von Willebrand factor (vWF) and platelet adhesion.

Proteins in the blood that are essential for coagulation, working in a complex cascade to form a clot. Notable ones include Factor VIII, IX, and X.

The process that prevents blood clots from growing and becoming problematic, primarily through the action of the enzyme plasmin which degrades fibrin.