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Joint stiffness, limited range of motion, and deformity due to glycated end products in collagen.

Reduced blood flow to extremities leading to pain, potential for osteonecrosis and amputation risks.

Increased pressure on foot joints leading to ulcers and potential amputations due to neuropathy and poor circulation.

Shoulder stiffness and pain due to inflammation and fibrotic adhesions, with a higher incidence in diabetics.

Joint damage and bone fractures due to loss of sensation from peripheral neuropathy.

Increased risk of osteomyelitis and chronic wounds due to poor circulation and immune response.

Compression of the median nerve in the wrist can be more prevalent in diabetic patients, leading to pain and numbness.

Accelerated cartilage degradation and joint pain exacerbated by obesity, which is common in type 2 diabetes.

Progressive degeneration of a weight-bearing joint, leading to fractures and deformities, often due to neuropathy.

Calcification and hardening of ligaments in the spine resulting in decreased range of motion; linked with insulin resistance.

Increased risk of fractures and bone density loss due to insulin deficiency affecting bone turnover.

Higher susceptibility to bone and joint infections due to immune system impairment, including septic arthritis.

Causes tight, inflexible joints, particularly in the hands, which can affect the patient's ability to perform daily tasks.

Fibrous tissue forms in the palm and fingers, causing finger contractures, more frequently seen in diabetic individuals.

Bone marrow inflammation that can cause severe pain and is sometimes found in diabetic patients.

Heel pain and inflammation of the plantar fascia may be more common in diabetics due to changes in weight distribution.

Muscle weakness and wasting due to acute muscle infarction, commonly seen in poorly controlled diabetes.