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Joint stiffness, limited range of motion, and deformity due to glycated end products in collagen.

Increased risk of fractures and bone density loss due to insulin deficiency affecting bone turnover.

Accelerated cartilage degradation and joint pain exacerbated by obesity, which is common in type 2 diabetes.

Progressive degeneration of a weight-bearing joint, leading to fractures and deformities, often due to neuropathy.

Muscle weakness and wasting due to acute muscle infarction, commonly seen in poorly controlled diabetes.

Increased risk of osteomyelitis and chronic wounds due to poor circulation and immune response.

Shoulder stiffness and pain due to inflammation and fibrotic adhesions, with a higher incidence in diabetics.

Fibrous tissue forms in the palm and fingers, causing finger contractures, more frequently seen in diabetic individuals.

Increased pressure on foot joints leading to ulcers and potential amputations due to neuropathy and poor circulation.

Tendons may thicken and become less flexible, causing movement restriction due to glycation.

Calcification and hardening of ligaments in the spine resulting in decreased range of motion; linked with insulin resistance.

Reduced blood flow to extremities leading to pain, potential for osteonecrosis and amputation risks.

Higher susceptibility to bone and joint infections due to immune system impairment, including septic arthritis.

Joint damage and bone fractures due to loss of sensation from peripheral neuropathy.

Heel pain and inflammation of the plantar fascia may be more common in diabetics due to changes in weight distribution.

Causes tight, inflexible joints, particularly in the hands, which can affect the patient's ability to perform daily tasks.

Compression of the median nerve in the wrist can be more prevalent in diabetic patients, leading to pain and numbness.