Discover published sets by community

Indicated by a broad, notched P wave in lead II and a biphasic P wave in V1; suggests increased left atrial pressure or volume.

Suggests thickening of the right ventricle; characterized by a tall R wave in V1 and a deep S wave in V6.

Evidenced by tall, peaked P waves in lead II and a biphasic P wave with a prominent initial component in V1; may indicate pulmonary disease or tricuspid valve disease.

Regular beat-to-beat variations in the amplitude of QRS complexes; often associated with pericardial effusion.

Reflects the total time for ventricular depolarization and repolarization; prolonged QT can lead to torsades de pointes.

Wave following the T wave; its presence can be associated with hypokalemia or bradycardia.

A heartbeat that occurs earlier than expected and originates from the atria; can be benign or indicate underlying pathology.

Time from the onset of the P wave to the start of the QRS complex; prolonged PR interval may indicate first-degree heart block.

Normal rhythm of the heart; each P wave is followed by a QRS complex and is originated from the sinoatrial node.

Rapid heart rate due to an atrial reentrant circuit; characterized by a 'sawtooth' pattern in the P waves, typically in the inferior leads.

Some P waves do not lead to a QRS complex despite a constant PR interval; may progress to a third-degree block.

Inflammation of the pericardium; ECG shows diffuse ST segment elevation without reciprocal changes and PR segment depression.

Represents ventricular depolarization; widened QRS may signify bundle branch block or ventricular hypertrophy.

A rapid, regular heartbeat that arises from improper electrical activity in ventricular tissue; may lead to ventricular fibrillation.

Delay or block in the conduction pathway of the right or left bundle branches; results in wide QRS complexes (>120 milliseconds).

Slowed conduction without missed beats; the PR interval is prolonged (>200 milliseconds).

Progressively lengthening PR interval until a beat is dropped; not every P wave is followed by a QRS.

High potassium levels that can cause peaked T waves, widened QRS complexes, and in severe cases, can lead to cardiac arrest.

Indicates ventricular repolarization; abnormal T waves can signify ischemia, hyperkalemia, or left ventricular hypertrophy.

A heartbeat that occurs earlier than expected and originates from the ventricles; frequent PVCs may require further evaluation.

Low potassium levels that may lead to flat T waves, prominent U waves, and potentially arrhythmias.

Segment between the QRS complex and T wave; elevation or depression may indicate myocardial infarction or ischemia.

The baseline of the ECG; deviations may indicate electrical injury or artifact.

A slower-than-normal heart rate; can indicate high vagal tone, inherent sinus node dysfunction, or be normal in athletes.

Disorganized electrical signals that cause inefficient heart contractions; can lead to sudden cardiac death.

Irregular, often rapid heart rate causing poor blood flow; characterized by absence of P waves and irregularly irregular rhythm.

No atrial impulses pass to the ventricles, leading to independent atrial and ventricular rhythms; typically requires a pacemaker.

Suggests thickening of the left ventricle; often demonstrates a high R wave in lateral leads and a deep S wave in the right precordial leads.

Represents atrial depolarization; can indicate atrial enlargement if abnormal.

A faster-than-normal heart rate; may result from fever, anxiety, hypovolemia, or heart failure.